Israel Hanukoglu, Ph. D.
Laboratory of Cell Biology
Ariel University
Ariel 40700, Israel

International Collaborations of our Laboratory

Throughout the years we have been blessed with a series of outstanding collaborations with the following laboratories from around the world.

CountryLab. HeadCo-workersProjectPubl.*
CanadaJ.-G. LeHouxACTH regulation of gene expression in adrenal cortex.66
GermanyGeorg E. SchulzG. Ziegler
C. Vonrhein
Expression and determination of the crystal structure of adrenodoxin reductase.58, 59, 60, 61
IsraelAaron HanukogluAdrenal steroidogenesis.27, 45
Aldosterone unresponsiveness (pseudohypoaldosteronism, PHA) due to Epithelial Sodium Channel (ENaC) mutations.46, 50, 51, 53, 56, 62-65, 67, 69-75, 77
IsraelA. AmsterdamB.S. SuhExpression of mitochondrial P450 system enzymes in ovarian granulosa cell lines.26, 33
IsraelNathan DascalFunctional expression of ENaC in Xenopus oocytes.69, 71, 74, 77
IsraelFortune KohenLinet LeviCloning of a novel cDNA from a rat pituitary library.38
IsraelDoron LancetP. NefCloning of the olfactory tissue specific P450.23
IsraelDavid SklanRoles of antioxidants in absorbing free radicals produced by mitochondrial P450 systems.40, 42, 43
IsraelMichael SteinitzProduction of cloned cell lines from lymphoblasts of PHA patients.53
ItalyEnzo OttavianiA. FranchiniExpression of ACTH receptor mRNA in human and molluscan immunoocytes.55
Expression of Epithelial Sodium Channel (ENaC) subunits in invertebrates.64
TurkeyEmel ArincA. SenAnalysis of the tissue specific expression of a microsomal cytochrome P450 in sheep.47
U.K.R. M. Gardiner S.S. Strautnieks
R.J. Thompson
Microsatellite based gene mapping to identity the genetic locus of pseudohypoaldosteronism mutations and sharing of patient pools for a multi-national study of PHA mutations.46, 50, 53, 56
U.S.A.Richard P. LiftonS.S. ChangIdentification of mutations in sodium channel subunits of PHA patients.51
U.S.A.Walter L. MillerS.B. Solish Cloning the human cDNA for adrenodoxin reductase.21
U.S.A.John E. ShivelyM. HaniuDetermination of amino acid sequences of adrenodoxin reductase fragments.20

* The numbers refer to the list of publications